Alterações arteriais na reação anafilática por picada de abelha: uma abordagem clínica

Authors

  • Lara Santos de souza Faculdade Rebouças de Campina grande
  • Maria Clara Ouriques Nascimento Faculdade Rebouças de Campina Grande
  • Carlos Andrey Duarte Da Silva Faculdade Rebouças de Campina Grande
  • Maria Luiza dos Santos Neta Faculdade Rebouças de Campina Grande
  • Rebeca Martins Pinto Faculdade Rebouças de Campina Grande
  • Thyago Araújo Gurjão Faculdade Rebouças de Campina Grande
  • Francisco de Assys Romero da Mota Sousa Faculdade Rebouças de Campina Grande
  • Larissa Silva Nelo Oliveira Faculdade Rebouças de Campina Grande
  • Nágela Maria Henrique Mascarenhas Faculdade Rebouças de Campina Grande

Keywords:

Anafilaxia, Histamina, Hipotensão, Vasodilatação, Epinefrina

Abstract

 

 

The anaphylactic reaction caused by a bee sting can result in significant arterial changes, affecting the body's circulatory dynamics and homeostasis. The toxin released by the bee is responsible for the action of proteins capable of provoking an intense immune response in sensitized individuals. This mechanism triggers an immediate allergic reaction, marked by the substantial release of inflammatory mediators such as histamine and leukotrienes into the bloodstream. The cardiovascular system is significantly influenced by these mediators. Histamine, for example, induces the dilation of blood vessels (vasodilation) and increases vascular permeability. This dilation of the vessels results in a decrease in peripheral vascular resistance, potentially causing a sudden decrease in blood pressure (hypotension). Increased permeability allows fluids to accumulate in the tissues, leading to edema and exacerbating feelings of shortness of breath and swelling in areas such as the throat and tongue. In addition, cardiac function and peripheral circulation can be impacted by an anaphylactic reaction. If not treated promptly, low blood pressure can decrease blood flow to essential organs, potentially resulting in anaphylactic shock. The cardiovascular response may also manifest as tachycardia (an elevated heart rate), which serves as a compensatory mechanism for the heart to maintain cardiac output despite the drop in blood pressure. Treatment of anaphylaxis requires immediate administration of epinephrine, a substance that functions as a vasoconstrictor, helping to reverse vasodilation and increase blood pressure. In addition, epinephrine decreases vascular permeability and relieves airway constriction. Additional management may include intravenous fluids, corticosteroids, and antihistamines to control the allergic reaction and prevent recurrence.

 

Published

2024-09-07

How to Cite

Santos de souza, L., Clara Ouriques Nascimento, M., Andrey Duarte Da Silva, C., Luiza dos Santos Neta, M., Martins Pinto, R., Araújo Gurjão, T., de Assys Romero da Mota Sousa, F., Silva Nelo Oliveira, L., & Maria Henrique Mascarenhas, N. (2024). Alterações arteriais na reação anafilática por picada de abelha: uma abordagem clínica. Caderno Verde De Agroecologia E Desenvolvimento Sustentável, 13(2), 19–19. Retrieved from https://gvaa.com.br/revista/index.php/CVADS/article/view/10917

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